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It was reported in 2002 that tibolone or its metabolite δ 4 -tibolone is transformed by aromatase into the potent estrogen 7α-methylethinylestradiol in women, analogously to the transformation of norethisterone into ethinylestradiol .  Controversy and disagreement followed when other researchers were unable to reproduce the findings however.       By 2008, it was established that tibolone is not aromatized in women and that the previous findings of 7α-methylethinylestradiol were merely a methdological artifact.    In accordance, a 2009 study found that an aromatase inhibitor had no effect on the estrogenic potencies of tibolone or its metabolites in vitro , unlike the case of testosterone .  In addition, another 2009 study found that the estrogenic effects of tibolone on adiposity in rats do not require aromatization (as indicated by the use of aromatase knockout mice ), further in support that 3α-hydroxytibolone and 3β-hydroxytibolone are indeed responsible for such effects.